DIATHESIS-STRESS MODEL - Psychology Dictionary

What Is the Diathesis Stress Model? | LoveToKnow

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This finding provided evidence for the Beckian version of the diathesis-stress hypothesis�." (Lewinsohn et al., 2001, p.210) An interesting thing to note is that their positive findings for risk factor support Beck's idea that early experience leads to the formation of dysfunctional beliefs, which other studies have not been able to show.

Psychology Definition of DIATHESIS-STRESS MODEL: Theory stating that mental and physical disorders develop from genetic or biological predisposition combined with stress.

Diathesis–stress model was nominated ..

It is widespread public opinion that so-called rational suicides exist, and it is subject to much debate [Hendin, 1998; Herrestad and Mehlum, 2005]. The term rational can be used to indicate that the suicidal person is not mentally disturbed and has considered suicide very carefully for a long period. It is tempting to conclude that the term also implies that doctors or others agree with the suicidal person in his or her pessimistic evaluation of future possibilities. The term rational is loaded with the illusion that there is nothing to gain from further exploration. However, the clinical world is full of examples of how the determined suicidal person can change his or her mind after finding relief from physical or psychological pain, after coming to terms with sadly changed perspectives, and after having received treatment of depressive symptoms expressed as long lasting hopelessness. The diathesis-stress model can be used as a framework for also understanding so-called rational suicides, and it opens the possibility for reconsidering the stressful situation and the person's ability to cope with this situation.

The diathesis-stress hypothesis of schizophrenia states that biological factors put someone at risk of schizophrenia, but environmental stressors are - 7610275

In describing the conceptual basis of a stress intervention method, Emotional Brain Training (EBT), a program which integrates advances in neuroscience and stress physiology, we propose a new paradigm for health care. Many health care treatments focus on managing symptoms of stress-related disorders. In modern society stress is primarily psychological in nature and in its chronic form, the result of (non-homeostatic) neural circuits that amplify and prolong stress. The result is cognitive, emotional, behavioral and physiologic dysregulation resulting in wear and tear (allostatic load). The effectiveness of treating any one stress symptom is likely decreased because of the persistent allostatic state. Emerging understanding of neuroplasticity suggests that this circuitry is capable of change. EBT is based on the repeated use of techniques that mirror secure attachment and optimal self-regulatory processing to alter allostatic circuits through the process of reconsolidation, therefore decreasing allostatic load. This results in an improved state of well-being. We hypothesize that decreased dominance of allostatic neuronal circuits leads to improved health outcomes, offering a new paradigm for health care.

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